嗜肺軍團菌尿液檢測試紙

廣州健侖生物科技有限公司

（廣州健侖生物科技有限公司是集研制開發(fā)、銷售、服務(wù)于一體的優(yōu)良企業(yè),公司產(chǎn)品涉及臨床快速診斷試劑、食品安全檢測試劑，違禁品快速檢測，動物疾病防疫檢測試劑，免疫診斷試劑、臨床血液學和體液學檢驗試劑、微生物檢驗試劑、分子生物學檢驗試劑、臨床生化試劑、有機試劑等眾多領(lǐng)域，同時核心代理Panbio、FOCUS、Qiagen、IBL、CORTEZ、Fuller、Inbios、BinaxNOW、LumuQuick、日本富士、日本生研等多家有名診斷產(chǎn)品集團公司產(chǎn)品，致力于為商檢單位、疾病預防控制中心、海關(guān)出入境檢疫局、衛(wèi)生防疫單位,緝毒系統(tǒng)，戒毒中心，檢驗檢疫單位、生化企業(yè)、科研院所、醫(yī)療機構(gòu)等機構(gòu)與行業(yè)提供*、高品質(zhì)的產(chǎn)品服務(wù)。此外，本公司還開展食品、衛(wèi)生、環(huán)境、藥品等多方面的第三方檢測服務(wù)。）

主要用途：用于檢測尿樣中嗜肺軍團菌血清型1抗原，以支持軍團菌感染的診斷。

產(chǎn)品規(guī)格：20T/盒

存儲條件：2-30℃

嗜肺軍團菌尿液檢測試紙

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【公司名稱】 廣州健侖生物科技有限公司
【】    楊永漢 
【】 
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【公司地址】 廣州清華科技園創(chuàng)新基地番禺石樓鎮(zhèn)創(chuàng)啟路63號二期2幢101-3室
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6.攝取與分泌神經(jīng)遞質(zhì)
神經(jīng)膠質(zhì)細胞既能攝取，又能分泌神經(jīng)遞質(zhì)。如：腦內(nèi)星形膠質(zhì)細胞能攝取谷氨酸與γ－氨基丁酸兩種遞質(zhì)，以消除這兩種遞質(zhì)對神經(jīng)元的持續(xù)作用；同時又可通過星形膠質(zhì)細胞的代謝，將這兩種遞質(zhì)再轉(zhuǎn)變?yōu)樯窠?jīng)元可重新利用的遞質(zhì)前體物質(zhì)。此外，星形膠質(zhì)細胞還能合成并分泌血管緊張素原、前列腺素、白細胞介素以及多種神經(jīng)營養(yǎng)病毒子等生物活性物質(zhì)。神經(jīng)膠質(zhì)細胞通過對神經(jīng)遞質(zhì)或生物活性物質(zhì)的攝取、合成與分泌。而發(fā)揮其對神經(jīng)元功能活動的調(diào)節(jié)作用。
7.吞噬作用
此外，小膠質(zhì)細胞和星形膠質(zhì)細胞有吞噬作用，能吞噬有病變的神經(jīng)
1. 神經(jīng)元急性壞死（紅色神經(jīng)元，red neuron）：急性缺血、缺氧，急性中毒或感染可引起神經(jīng)元的死亡，表現(xiàn)為神經(jīng)元核固縮，胞體縮小變形，胞漿尼氏小體（Nissl body）消病毒，HE染色胞漿呈深伊紅色，稱為紅色神經(jīng)元(red neuron)。如細胞壞死后的酶性分解過程繼續(xù)發(fā)展，則可導致細胞核溶解消病毒，殘留細胞的輪廓或痕跡稱為鬼影細胞（ghost cell）。由缺血引起的紅色神經(jīng)元zui常見于大腦皮質(zhì)的錐體細胞和小腦蒲肯野（Purkinje）細胞。
2. 單純性神經(jīng)元萎縮（simple neuronal atrophy）:神經(jīng)元慢性漸進性變性以至死亡的過程稱為單純性神經(jīng)元 萎縮。又稱神經(jīng)元的慢性病變。病變特點表現(xiàn)為神經(jīng)元胞體縮小，核固縮而無明顯的尼氏小體溶解，一般 不伴炎癥反應(yīng)。晚期可伴明顯膠質(zhì)細胞增生。
3. 中央性Nissl小體溶解（ce病毒al chromatolysis）：為一種可逆性變性，常由病毒感染，維生素B缺乏及神經(jīng) 元與軸突斷離等病毒素所致。病變表現(xiàn)為神經(jīng)元腫脹、變圓、核偏位，胞漿中央的尼氏小體崩解，進而溶解 消病毒，或僅在細胞周邊部有少量殘余，胞漿著色淺而呈蒼白均質(zhì)狀。
4. 神經(jīng)元胞質(zhì)內(nèi)包含體形成（i病毒ancyto-plasmic inclusion）：神經(jīng)元胞質(zhì)內(nèi)包含體形成可見于某些病毒感染 和變性疾病等，其形態(tài)、大小和著色不同，分布部位也有一定規(guī)律，如Parkinson病的黑質(zhì)，藍斑等處的神 經(jīng)細胞中的Lewy小體（圖14-2）；狂犬病時海馬和腦皮質(zhì)錐體細胞中的Negri小體，分別對這些疾病具有診 斷意義。此外，神經(jīng)元胞漿中出現(xiàn)脂褐素多見于老年人。

6. Uptake and secretion of neurotransmitters
Glial cells can not only uptake, but also neurotransmitters. Such as: astrocytes in the brain can take up glutamate and γ-aminobutyric acid two neurotransmitters in order to eliminate the two neurotransmitters on the sustained effect; at the same time through the astrocyte metabolism , Converting these two neurotransmitters into neurotransmitter precursors. In addition, astrocytes also synthesize and secrete biologically active substances such as angiotensinogen, prostaglandins, interleukins and various neurotrophic viruses. Glial cells through the neurotransmitter or bioactive substances uptake, synthesis and secretion. And play its role in the regulation of neuronal functional activity.
7. phagocytosis
In addition, microglia and astrocytes have phagocytosis that engulfs diseased nerves
1. Neuronal Acute Necrosis (red neuron): Acute ischemia, hypoxia, acute poisoning or infection can cause neuronal death, manifested as neuron nuclear condensation, cell body deformation, cytoplasmic Nissl small Body (Nissl body) virus, HE staining cytoplasm dark red, known as red neurons (red neuron). If the process of enzymatic decomposition after necrosis continues, the nuclei will dissolve the virus and the outline or trace of residual cells will be called the ghost cell. Red neurons caused by ischemia are most commonly found in the pyramidal cells of the cerebral cortex and in Purkinje cells of the cerebellum.
2. Simple atrophy of nerve cells (simple neuronal atrophy): The process of chronic progressive degeneration and death of neurons is called simple neuronal atrophy. Also known as neuronal chronic disease. Lesions characterized by shrinkage of neuronal somatic cells, nuclear pyknosis and no significant Nissl body lysis, generally do not have inflammatory reactions. Late with obvious glial cell proliferation.
3. Central Nissl body lysis (ce virus al chromatolysis): a reversible degeneration, often caused by viral infection, vitamin B deficiency and neurons and axons off the other toxins. Lesions showed neuronal swelling, rounding, nuclear deviation, the central cytoplasmic Nissl body disintegration, and then dissolve the virus, or only a small amount of residual cells in the periphery, the pale cytoplasm was pale homogeneous shape .
4. Neuronal cytoplasmic inclusion body formation (i virus ancyto-plasmic inclusion): Neuronal cytoplasmic inclusion bodies can be seen in some viral infections and degenerative diseases, the shape, size and coloring of different distribution sites also have a Such as Lewy bodies in neurons at the substantia nigra and locus coeruleus of Parkinson's disease (Figure 14-2); Negri bodies in the hippocampus and pyramidal cells of rabbits at the time of rabies are diagnostic for these diseases, respectively . In addition, the presence of lipofuscin in neuronal cytoplasm is more common in the elderly.